Wednesday, November 4, 2009

Question for the masses

A question has been raised in my cardiology class that no one seems to be able to answer to the student's satisfaction.

What is happening, at the cellular level, that causes ST segment changes during an MI? I know it has something to do with repolarization changes due to dead and dying or ischemic tissue, but she wants to know the specific reasons behind the amplitude changes.

Any takers among my 2 or 3 readers?

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